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人體副腎 特히 皮質의 病理組織學的 硏究

Pathologic Histologic Studies on Human Adrenal Gland especially on Adrenal Cortex

중앙의학 1962년 3권 4호 p.357 ~ 373
강처일,
소속 상세정보
강처일 (  ) - Seoul National University Medical Faculty Department of Pathology

Abstract


The author reviewed the inter-relation-ship between the fat content of the adrenal cortex and the development of fat tissue of the body, and observed, in detail, the development of fat in the adrenal cortex and the development of the adrenal cortex.
Teh author used, as standards, the findings if adrenal cortices of 5 cases of caseualty death which were assumed to be most normal.
The adrenals of 100 necropsies cases with death due to various diseases were studied and the following finding observed.
1. Fat decrease in the adreal cortex may in 3 ways.
a) diffuse decrease of fat throughout the zona glomerulosa, zona fasciculata and zona reticularis.
b)Systematic decrease of fat in one of the 3 adreno-cortical layers, or
c) patchy decrease of fat anywhere in the adrenal cortex.
2. in general the total fat content of the adrenal cortex revealed by Kawamura Yazaki Sudan Ⅲ mehtod and lipoid substances stained by Smith-Dictrich method increase or decrease in parallel, though at times there is no parallel.
3. Case of chronic mephritis show a menarkable amount of fat in the adrenal cortex, particularly lipoid substance.
4. The fat content in the adrenal cortex parallels in general the degree of development of fat tissue of the body. The cortical fat decreases markedly in cases of cachexia.
5. The suthor messured the thickness as an index of the development of adrenal cortex.
The ratio in thickness of the zona glomerulosa, zona fasxixulata and zona reticularis is roughly 10: 69: 21.
The increase in thickness of the adrenal cortex is mainly the result of hypertrophy of zona fasciculata and secondly zona reticullaris.
Decrease in thickness of sdrenal cortex is mainly the result of atrophy of zona fasciculata.
6. It is considered in corrext to estimate the hyper-or hypo-function of the adrenal cortex by only increase or decrease of the adrenal cortical tissue.
The histolgical finding of the cases with marked increase of adrenal cortex include hyperemia, hemorrhage, retrogressive, degeneration of cortical cells, necrosis, edema or marked decrease of fat substance indicating injury of various kinds to this organ and presumably indicate lowering of functional activity of this organ. Conversely the adrenal cortical tissue in cases with thin cortex is not necessarily atrophic in nature and fat deposition may actually by increased.

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